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This emerging relationship data had been presented by Dr. Han and researchers at Stanford in 2017, with their initial research from the institution reporting the need to increase focus and control groups for EBV-specific data for the MS population. Jump ahead to the Bjornevik et al. (2022)<\/a> study out of Harvard which has now been momentous for identifying causative statistics with robust sample sizes utilizing thousands of military personnel for data. Of the 801 reported service members who developed MS during their 10-year study period, only one was identified not to have the presence of the Epstein-Barr virus (<\/a>Bjornevik et al., 2022<\/a>)<\/a>. This study, among others, is building off several historical investigations and theories of EBV being a significant factor in the triggering of MS and destruction of the myelin sheath in patients.<\/p>\n Additional groundbreaking results this year from both universities are more significantly tying this relationship of EBV to trigger MS effects and disease development. Lanz et al. (2022)<\/a> deduced that the nature of how EBV binds to GlialCAM triggers the destruction of the myelin sheath by the body\u2019s own response to fight the virus. Being able to clearly identify this destructive force paves the way for more focus on treatments and possible vaccines to address this devastating response within the body. Returning to Harvard University\u2019s study by Bjornevik et al. (2022)<\/a> suggests that interventions can now be further developed to better treat EBV as well as the presentation of MS in patients in clinical management.<\/p>\n <\/p>\n![\"\"](\"http:\/\/l_new.local\/wp-content\/uploads\/CV-brain.jpg\")
<\/a>Let us honor Multiple Sclerosis Awareness Month by celebrating some recent breakthroughs in MS research. The linking and investigation of a connection between the Epstein-Barr Virus (EBV) and MS have been developing for the past several years. However, recent publications from Harvard and Stanford University researchers have profoundly impacted this cause-and-effect relationship to better identify at-risk populations and provide guidance for further development of treatments to promote functional longevity in our MS patients.<\/p>\n